OBESITY TREATMENT FOR PATIENTS WITH CARDIOVASCULAR DISEASE
Obesity significantly increases the risk of both arterial
and venous diseases. The incidence of coronary heart disease (CHD), congestive
heart failure (CHF), stroke, atrial fibrillation (AF), and deep venous
thrombosis (DVT) increases with increased BMI or weight. Our daily excess
carbohydrate intake enhances inflammation and hastens the onset of these
diseases.
Bad lipids, insulin resistance (IR), and CHF are further
linked through the concept of “lipotoxicity.” This suggests that triglyceride accumulation
in the liver (non-alcoholic fatty liver disease-NAFLD), in muscle (especially
heart muscle) and the pancreatic beta cells (insulin makers) lead to IR in the
liver, decreased heart function, and loss of beta cell insulin release. MRI
studies have demonstrated dysfunction in heart muscle that resulted in CHF.
Increased AF incidence is associated with obesity based on obstructive sleep
apnea (OSA) with low oxygen in the blood and increased blood pressure in the
lungs.
Weight loss induced by a VLCD (Very Low Carbohydrate Diet)
with onsite MD supervision, has shown to be very effective in improving heart
function. After only 4 weeks on a VLCD there is a reduction in fat around the
heart and the heart pumps better.
Another study by ultrasound demonstrated increased heart function in 4
weeks.
Patients on a VLCD with Coumadin need frequent monitoring
and the other anti-coagulants do not.
Weight loss will also lessen the risk for initial or
recurrent lower extremity DVTs, improve peripheral arterial function, and
reduce atrial fibrillation risk all by reducing obstructive sleep apnea.
Remember sometimes this goes unnoticed for years and needs to be evaluated by
the MD supervising onsite the VLCD.
Ketosis the result of VLCD will cause excess water to be
cleared by urination. Thus sodium restriction is achieved. Are you ready to
help fix your body? Start now.
Dr. B
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